Overexpression of CuZn-SOD Prevents Lipopolysaccharide-Induced Endothelial Dysfunction

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Overexpression of CuZn-SOD prevents lipopolysaccharide-induced endothelial dysfunction.

BACKGROUND AND PURPOSE Inflammation is thought to be a major contributor to carotid artery disease. Lipopolysaccharide (LPS) activates inflammatory mechanisms thought to contribute to endothelial dysfunction by mechanisms that are not well defined. The goal of this study was to determine whether overexpression of CuZn-SOD protects against LPS-induced increases in superoxide and endothelial dysf...

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Ceramide-induced impairment of endothelial function is prevented by CuZn superoxide dismutase overexpression.

OBJECTIVE Ceramide is an important intracellular second messenger that may also increase superoxide. The goal of this study was to determine whether overexpression of CuZn superoxide dismutase (SOD) protects against ceramide-induced increases in vascular superoxide and endothelial dysfunction. METHODS AND RESULTS Carotid arteries from CuZnSOD-transgenic (CuZnSOD-Tg) and nontransgenic litterma...

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Gene transfer of Cu/Zn SOD to cerebral vessels prevents FPI-induced CBF autoregulatory dysfunction.

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The goal of the present study was to test the hypothesis that the CuZn isoform of superoxide dismutase (CuZnSOD) protects against angiotensin II (Ang II)-induced endothelial dysfunction. Vascular responses of carotid arteries from control, CuZnSOD-deficient (CuZnSOD(+/-)), and CuZnSOD transgenic mice were examined in vitro after overnight incubation with either vehicle or Ang II (1 or 10 nmol/L...

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ژورنال

عنوان ژورنال: Stroke

سال: 2004

ISSN: 0039-2499,1524-4628

DOI: 10.1161/01.str.0000132764.06878.c5